Cirrhosis

  • Irreversible liver damage
  • Histologically there is loss or normal hepatic architecture with fibrosis and nodular regeneration

Causes

  • Alcohol
  • HBV or HCV infection
  • Autoimmune disorders - Primary biliary cirrhosis, primary sclerosising cholangitis, autoimmune hepatitis
  • Genetic disorders - ?1-antitrypsin deficiency, haemachromatosis, Wilson’s disease
  • Budd-Chiari syndrome
  • Drugs - amiodarone, methydopa, methotrexate

Signs

  • See liver failure

Complications

  • Hepatic failure
    • Coagulopathy
    • Encephalopathy
    • Hypoalbuminaemia
    • Sepsis
    • Spontaneous bacterial peritonitis
    • Hypoglycaemia
  • Portal hypertension
    • Ascites
    • Splenomegaly
    • Portosystemic shunting

Tests

  • Blood
    • LFTs - bilirubin, AST, ALT, alk phos, ?-GT
    • Reduced albumin, increased PT/INR
    • Reduced WCC and platelets indicates hypersplenism
  • Finding the cause;
    • Ferritin
    • Iron/total iron binding capacity
    • Hepatitis serology
    • Immunoglobulins (IgM)
    • Autoantibodies (ANA, AMA, SMA)
    • ?-fetoprotein
    • Caeruloplasmin in patients <40
    • ?1-antitrypsin

 

Management

  • Good nutrition - low salt diet, alcohol abstinence
  • Avoid NSAIDs, opiates and sedatives
  • Cholestyramine for puritis
  • Consider USS and ?-fetoprotein to screen for HCC every 6mth
  • Specifically;
    • Interferron- ?  improves LFTs and may delay onset of HCC in HCV cirrhosis
    • High dose ursodeoxycholic acid in PBC (normalises LFTs but doenst affect progression)
    • Penecillamine in Wilson’s disease
    • Ascites - fluid and salt restrict. Give spironolactone and if this doesn’t work add frusemide
    • SBP - common organisms are E.coli, Klebsiella and Streptococcus. Give cefotaxime for 5d or until sensitivities are known. Consider prophylaxis in high risk patient or who have had a previous episode (norfloxacin)

Prognosis

  • Overall 5 year survival is 50%
  • Poor prognostic factors are - encephalitis, serum Na <110, serum albumin <25 and increased INR

Child-Pugh grading of cirrhosis

  • Based on;
    • Bilirubin
    • Albumin
    • Prothrombin time
    • Ascites
    • Encephalopathy
  • Grading can be used to predict prognosis and the quantify need for transplantation
  • If the score is >8 there is increased risk of variceal bleeding

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Disclaimer: These notes are my own personal study aid - DO NOT use them for medical advice!