Gynaecological infections

  • If one STI is present, there may be others

 

Lower Genital Tract

  • Prior to puberty, the vaginal epithelium is cuboidal and the pH is neutral
  • After puberty under the influence of oestrogen, stratified squamous epithelium develops and lactobacilli become the predominant commensals. This results in a drop in vaginal pH to 3.5-4.5
  • After menopause the vaginal epithelium become atrophic and the bacterial flora becomes similar to that of the skin. pH rise to neutral again

 

Physiological discharge

  • Normally white, turns yellow with oxidation
  • Consists of desquamated epithelium, mucus, bacteria and vaginal transudate fluid
  • Discharge increases midcycle due to increased mucus production
  • Also increases during pregnancy and sometimes if the woman is on the contraceptive pill

 

Vaginal Candidiasis

  • Over ¾ of women have had at least one episode
  • Opportunistic infection, not an STI
  • Presents are itching and soreness of the vagina and vulva with a white, curdy, yeasty smelling discharge
  • pH of vagina is normal
  • Predisposing factors
    • Immunosuppression
    • Diabetes
    • Increased oestrogen
    • Pregnancy
    • High dose oral contraceptive pill
    • Broad-spectrum antibiotic therapy
    • Vaginal douching, tight fitting clothes etc.
  • Treatment
    • Topical clotrimazole 500mg
    • Oral fluconazole 150mg (only affective against candida albican – cause 80% of cases)

 

 

Bacterial Vaginosis

  • Commonest cause of abnormal discharge in women of childbearing age
  • Commoner in women of Afro-Caribbean origin or those with an IUD
  • Often arises spontaneously around the time of menstruation and then resolves spontaneously mid-cycle
  • Causes by anaerobic bacteria e.g. Gardnerella vaginalis, Bacteroides
  • Results in a rise in vaginal pH to between 4.5 and 7 and a disappearance or reduction in lactobacilli
  • Associated with an offensive fishy smelling discharge
  • Fishy smell due to the production of polyamines and trimethylamine by bacteria
  • Diagnostic criteria
    • Vaginal pH >4.5
    • Release of a fishy smell on addition of alkali
    • Characteristic discharge on examination
    • Presence of ‘clue cell’ on microscopy – vaginal epithelium cells which are so coated in bacteria that there borders are obscured
    • Cultures taken from high vaginal swabs – should see increase gram-positive and gram-negative cocci  in the absence of with reduced numbers of gram-positive bacilli
  • Treatment
    • Metronidazole
  • Women with BV are at increased risk of second trimester miscarriage and preterm delivery during pregnancy
  • Also at increased risk of infection following surgery
  • Treated women with metronidazole prior to termination reduces subsequent incidence of PID and endometriosis

 

 

Trichomoniasis

  • Caused by a protozoan parasite, Trichomonas vaginalis
  • STI which may be carried asymptomatically for several months prior to the development of symptoms
  • Often carried in men asymptomatically but may present as a urethritis
  • In women causes severe vulvovaginitis and a yellow/green purulent vaginal discharge
  • Punctuate haemorrhages occur on the cevix, ‘strawberry cervix’
  • Diagnosis is via culture of vaginal secretions. Causative organism has 4 flagella. PMNs may also be present
  • Treatment
    • Metronidazole
    • Sexual partners should also be treated

 

 

Upper genital tract infections

  • 80% of PID caused by gonorrhoea and Chlamydia
  • PID results in tubule damage leading to ectopic pregnancy and infertility as well as chronic pelvic pain

 

Non-specific cervicitis

  • Diagnosis is based on detecting purulent discharge at the cervical os
  • It is often accompanied by bleeding
  • Can be confused with a benign ectropion but this doesn’t generally bleed so readily
  • Symptoms include
    • Postcoital bleeding
    • Purulent vaginal discharge
  • Associated with;
    • Gonorrhoea
    • Chlamydia
    • Herpes simplex (ulceration present)
  • Nabothian follicles are mucus containing cysts which are often presnt following chronic cervicitis

 

Chlamydia trachomatis

  • Commonest STI in industrialised countries
  • Highest prevalence in women under 25
  • Asymptomic in 80% of women and 50% of men
  • In men it causes a urethritis
  • In women it causes cervicitis and PID
  • Intracellular bacterium, infects columnar epithelium in the genital tract
  • Samples must be taken from the endocervix and diagnosis is via ELISA or PCR
  • PCR can also be done on urine samples
  • Treatment
    • Doxycycline
    • Azithromycin
    • Ofloxacin
    • During pregnancy
      • Azithromycin
      • Erythromycin
  • Essential that sexual partners are also treated

 

Gonorrhoea

  • Prevalence of around 1% in women of child-bearing age
  • 50% of women are asymptomatic
  • 70% of men are symptomatic, symptoms include;
  • Urethritis
  • Green urethral discharge
  • Dysuria
  • Can be carried in the throat and cause exudative tonsillitis
  • Can also cause conjunctitis and proctitis
  • Neisseria gonorrhoeae is a gram-negative diplococcus which colonises the columnar epithelium of the genital tract
  • Diagnosis made by observing gram-negative diplococci, intracellularly or urethral, cervical and rectal swabs
  • Treatment – will depend on culture sensitivity. Mostly only requires a single dose
    • Amoxicillin
    • Ciprofloxacin
    • Spectinomycin
    • Azithromycin
    • Ceftriazome
    • Cefixime
  • Sexual partners must be also screened and treated
  • More than 50% of women with gonorrhoea also have Chlamydia, therefore treat together

 

Pelvic Inflammatory Disease

  • As infection ascends into the uterus endometritis develops which is associated with inter-menstrual bleeding
  • Salpingitis;
    • Mucosal inflammation, initially involving PMN, then mononuclear cells followed by plasma cells
    • Inflammatory exudates fills the tube and adhesions can develop
    • Inflammation extends over the serosal surface and pus exudes from the fimbriae over the ovaries and adnexae
    • Tubes can become fixed
    • Can result in pelvic peritonitis
    • Tubes can become blocked
    • Uterus can become fixed and retoverted
    • Hydrosalpinx is caused by accumulation of fluid within the tubes resulting in swelling
    • Pyosalpinx is infection within the tube
  • Clinical features
    • Pelvic pain
    • Deep dyspareunia
    • Intermenstrual bleeding
    • O/E cervical excitiation
    • Lower genitial tract infection, BV, trichomoniasis or cervicitis
    • Pyrexia, raised CRP and neutrophil count
    • Adnexal mass
    • Laproscopy is regarded as the gold standard for diagnosis
  • If PID is suspected, endocervical swabs should be taken for C. trachomatis and N. gonorrhoeae. A high vaginal swab should be taken for Trichomonas vaginalis and BV
  • Treatment – should cover both Chlamydia and gonorrheoea and well as anaerobics
    • Doxycycline and ciprofloxacin along with metronidazole or;
    • Ofloxacin with metronidazole
  • In patients who are systemically unwell;
    • I.V. cephalosporin and metronidazole along with a 2 week course of doxycycline

 

  • Intra-abdominal spread of Chlamydia and gonorrhoea can cause peri-appendicitis or peri-hepatitis
  • Peri-hepatitis is called Fits-Hugh Curtis syndrome
  • May present with right upper quadrant pain
  • May be misdiagnosed as cholecystitis
  • Disseminated infection with Chlamydia may cause Reiter’s syndrome or sexually acquired reactive arthritis
  • Occurs in less than 1% of cases
  • Asymmetrical oligoarthritis affecting the large joints of the lower limb
  • In Reiter’s syndrome there is also uveitis and rash
  • It is associated with HLA-B27
  • Disseminated infection with gonorrhoea is rare but can present as a septic oligoarthritis in the small joints of the wrist and hands. Also associated with a popular rash.

 

 

Genital Ulcer Disease

 

Herpes Simplex

  • Mostly associated with herpes simplex virus Type II
  • Present for life
  • Reports however that up to 50% of cases might be caused by Type I
  • May be subclinical for many years prior to presenting features developing
  • Diagnosis is made by extracting vesicular fluid from the ulcer and demonatrating viral particles via EM or culture on a tissue monolayer

 

Primary Herpes

  • Presents up to 3 weeks after infection
  • Widespread involvement of the vagina and vulva
  • Cervix may also be affected
  • Primary pharyngeal and rectal infections can also present
  • Painful vesicles can coalesce to form ulcers
  • Urinary retention can develop due to pain and involvement of the sacral nerves
  • Generally looks much worse than recurrent infection
  • Treatment
    • Bathing in salt water
    • Lignocaine local anaethetic gel
    • Antiviral treatment – acyclovir

 

Recurrent Herpes

  • Following a primary infection, herpes colonises the neurons of the dorsal root ganglion therefore establishing a latent infection
  • Recurrent disease can vary from asymptomatic shedding or virus to large ulcers which can develop if the patient is immunosuppressed
  • Antiviral treatment is usually ineffective at treating established recurrent infection
  • Advice is the bath in salt water and to avoid intercourse until vesicles have cleared up
  • Some individuals experience a prodrome prior to development of the vesicles. This may involve tingling or pain in the thigh or perineum
  • Some patients develop frequent reoccurrences which are quite debilitating. In these circumstances it might be appropriate to prescribe long term suppression  with acyclovir.

 

  • Virus can be transmitted in the absence of clinical signs it is therefore important that the patients uses condoms for intercourse

 

 

 

 

Syphilis

  • Systemic STI caused by Treponema pallidum
  • First manifestation is a painless ulcer (chancre) at the site of inoculation – in women most common on the cervix
  • Usually arises 3-6 weeks post infection
  • Usually associated with enlargement of the inguinal lymph nodes
  • Following the chancre, secondary syphilis can arise, this is associated with a non-itchy maculopapular rash mostly involving the palms and soles of the feet
  • Warts can be seen peri-anally
  • Mucus patches and linear ulcers can be seen on the mucosal surfaces
  • May be generalised lymphadenopathy
  • Sensorineural deafness can occur early in infection due to destruction of the inner ear hair cells
  • Alopecia, arthritis and meningitis can also develop
  • Diagnosis is made via microscopy and serological tests
  • Following resolution of secondary syphilis a period of latency occurs whereby there are no clinical manifestations of disease
  • Primary and secondary syphilis are not life threatening but late tertiary syphilis is
    • 5-10% can develop neurosyphilis
    • 20% will develop cardiovascular syphilis – aortic aneurism, aortic regurgitation
  • Risk of vertical transmission
  • This can cause intrauterine death
  • Prevention of vertical transmission requires mother to be treated before 20 weeks gestation
  • Treatment
    • Penicillin

 

 

Human papillomavirus

  • More than 70 different types have been decribed
  • Some strains  (6 and 11) can cause genital warts
  • These are sexually transmitted
  • Can affect the skin of the vulva, perineum, vagina, cervix and rectum
  • HPV 16 and 18 cause flat warts and have been linked to cervical carcinoma
  • Smoking is a strong risk factor
  • Treatment of genital warts
  • Cyotherapy
  • Application of podophyllin
  • Surgical treatment for intractable cases, laser, electrocautery or scissor excision

 

 

Molluscum contagiosum

  • Causes by pox virus
  • Painless pearly lesions up to 5mm in diameter
  • STI
  • Fluid from vesicles is infectious
  • Large confluent lesions can develop in patients who are immunosuppressed
  • Untreatable, cleared by own immune response
  • Can be treated with cyotherapy and application of phenol


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Disclaimer: These notes are my own personal study aid - DO NOT use them for medical advice!